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A fast growing number of literature indicates that MIF is implicated in the pathogenesis of sepsis, and inflammatory and autoimmune disorders, suggesting that MIF-directed therapies may possibly supply new therapy possibilities for human disorders Down the road.
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learned during the late sixties as an item of activated T cells, the cytokine macrophage migration inhibitory component (MIF) has been identified recently to carry out important features as a mediator from the innate immune program.
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The receptor that mediated MIF activity remained elusive until a examine in 2003, which utilized a cDNA library and fluorescently conjugated MIF to monitor for the receptor and discovered CD74 as the MIF receptor. The authors explained that MIF certain to the extracellular area of CD74, resulting in extracellular signal-controlled kinase (ERK) pathway activation (twenty five).